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Ischemic Heart Disease

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Articles published in
PLoS Genet
    April 2021
  1. HELKKULA P, Kiiskinen T, Havulinna AS, Karjalainen J, et al
    ANGPTL8 protein-truncating variant associated with lower serum triglycerides and risk of coronary disease.
    PLoS Genet. 2021;17:e1009501.
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    November 2020
  2. XUE H, Pan W
    Inferring causal direction between two traits in the presence of horizontal pleiotropy with GWAS summary data.
    PLoS Genet. 2020;16:e1009105.
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    April 2020
  3. EMDIN CA, Haas ME, Khera AV, Aragam K, et al
    A missense variant in Mitochondrial Amidoxime Reducing Component 1 gene and protection against liver disease.
    PLoS Genet. 2020;16:e1008629.
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    October 2019
  4. CENSIN JC, Peters SAE, Bovijn J, Ferreira T, et al
    Causal relationships between obesity and the leading causes of death in women and men.
    PLoS Genet. 2019;15:e1008405.
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    April 2019
  5. DAHL A, Cai N, Ko A, Laakso M, et al
    Reverse GWAS: Using genetics to identify and model phenotypic subtypes.
    PLoS Genet. 2019;15:e1008009.
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    December 2018
  6. BANERJEE S, Zeng L, Schunkert H, Soding J, et al
    Bayesian multiple logistic regression for case-control GWAS.
    PLoS Genet. 2018;14:e1007856.
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    November 2018
  7. NANDA V, Wang T, Pjanic M, Liu B, et al
    Functional regulatory mechanism of smooth muscle cell-restricted LMOD1 coronary artery disease locus.
    PLoS Genet. 2018;14:e1007755.
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    October 2018
  8. IYER D, Zhao Q, Wirka R, Naravane A, et al
    Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk.
    PLoS Genet. 2018;14:e1007681.
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    October 2017
  9. PATERNOSTER L, Tilling K, Davey Smith G
    Genetic epidemiology and Mendelian randomization for informing disease therapeutics: Conceptual and methodological challenges.
    PLoS Genet. 2017;13:e1006944.
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