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Articles published in
Cardiovasc Res
    June 2018
  1. LINDSEY ML
    Reg-ulating macrophage infiltration to alter wound healing following myocardial infarction.
    Cardiovasc Res. 2018 Jun 15. pii: 5038408. doi: 10.1093.
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    May 2018
  2. LORCHNER H, Hou Y, Adrian-Segarra JM, Kulhei J, et al
    Reg proteins direct accumulation of functionally distinct macrophage subsets after myocardial infarction.
    Cardiovasc Res. 2018 May 30. pii: 5025270. doi: 10.1093.
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    March 2018
  3. DEUTSCH MA, Doppler SA, Li X, Lahm H, et al
    Reactivation of the Nkx2.5 cardiac enhancer after myocardial infarction does not presage myogenesis.
    Cardiovasc Res. 2018 Mar 20. pii: 4944453. doi: 10.1093.
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  4. CHEN CW, Wang LL, Zaman S, Gordon J, et al
    Sustained Release of Endothelial Progenitor Cell-Derived Extracellular Vesicles from Shear-Thinning Hydrogels Improves Angiogenesis and Promotes Function after Myocardial Infarction.
    Cardiovasc Res. 2018 Mar 16. pii: 4939452. doi: 10.1093.
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    November 2017
  5. ZIEGLER KA, Ahles A, Wille T, Kerler J, et al
    Local sympathetic denervation attenuates myocardial inflammation and improves cardiac function after myocardial infarction in mice.
    Cardiovasc Res. 2017 Nov 24. pii: 4657089. doi: 10.1093.
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    September 2017
  6. PARK KC, Gaze DC, Collinson PO, Marber MS, et al
    Cardiac troponins: from myocardial infarction to chronic disease.
    Cardiovasc Res. 2017 Sep 14. doi: 10.1093.
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  7. HAUSENLOY DJ, Garcia-Dorado D, Erik Botker H, Davidson SM, et al
    Melatonin as a cardioprotective therapy following ST-segment elevation myocardial infarction: is it really promising? Reply.
    Cardiovasc Res. 2017;113:1418-1419.
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    July 2017
  8. BAYOUMI AS, Teoh JP, Aonuma T, Yuan Z, et al
    MicroRNA-532 protects the heart in acute myocardial infarction, and represses prss23, a positive regulator of endothelial-to-mesenchymal transition.
    Cardiovasc Res. 2017 Jul 11. doi: 10.1093.
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    June 2017
  9. SCHOBESBERGER S, Wright P, Tokar S, Bhargava A, et al
    T-tubule remodelling disturbs localized beta2-adrenergic signalling in rat ventricular myocytes during the progression of heart failure.
    Cardiovasc Res. 2017;113:770-782.
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  10. PERRINO C, Barabasi AL, Condorelli G, Davidson SM, et al
    Epigenomic and transcriptomic approaches in the post-genomic era: path to novel targets for diagnosis and therapy of the ischaemic heart? Position Paper of the European Society of Cardiology Working Group on Cellular Biology of the Heart.
    Cardiovasc Res. 2017;113:725-736.
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  11. MANSOR LS, Sousa Fialho MDL, Yea G, Coumans WA, et al
    Inhibition of sarcolemmal FAT/CD36 by sulfo-N-succinimidyl oleate rapidly corrects metabolism and restores function in the diabetic heart following hypoxia/reoxygenation.
    Cardiovasc Res. 2017;113:737-748.
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    May 2017
  12. HAUSENLOY DJ, Garcia-Dorado D, Botker HE, Davidson SM, et al
    Novel targets and future strategies for acute cardioprotection: Position Paper of the European Society of Cardiology Working Group on Cellular Biology of the Heart.
    Cardiovasc Res. 2017;113:564-585.
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  13. CHEN Z, Xie J, Hao H, Lin H, et al
    Ablation of periostin inhibits post-infarction myocardial regeneration in neonatal mice mediated by the phosphatidylinositol 3 kinase/glycogen synthase kinase 3beta/cyclin D1 signalling pathway.
    Cardiovasc Res. 2017;113:620-632.
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  14. VINCENT A, Sportouch C, Covinhes A, Barrere C, et al
    Cardiac mGluR1 metabotropic receptors in cardioprotection.
    Cardiovasc Res. 2017;113:644-655.
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  15. VALLE RALEIGH J, Mauro AG, Devarakonda T, Marchetti C, et al
    Reperfusion therapy with recombinant human relaxin-2 (Serelaxin) attenuates myocardial infarct size and NLRP3 inflammasome following ischemia/reperfusion injury via eNOS-dependent mechanism.
    Cardiovasc Res. 2017;113:609-619.
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    April 2017
  16. WANG Z, Huang S, Sheng Y, Peng X, et al
    Topiramate modulates post-infarction inflammation primarily by targeting monocytes or macrophages.
    Cardiovasc Res. 2017;113:475-487.
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    March 2017
  17. BROMAGE DI, Pickard JM, Rossello X, Ziff OJ, et al
    Remote ischaemic conditioning reduces infarct size in animal in vivo models of ischaemia-reperfusion injury: a systematic review and meta-analysis.
    Cardiovasc Res. 2017;113:288-297.
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    February 2017
  18. LIM S, Lee GY, Park HS, Lee DH, et al
    Attenuation of carotid neointimal formation after direct delivery of a recombinant adenovirus expressing glucagon-like peptide-1 in diabetic rats.
    Cardiovasc Res. 2017;113:183-194.
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    January 2017
  19. JOSE ALBURQUERQUE-BEJAR J, Barba I, Valls-Lacalle L, Ruiz-Meana M, et al
    Remote ischemic conditioning provides humoural cross-species cardioprotection through glycine receptor activation.
    Cardiovasc Res. 2017;113:52-60.
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  20. RAWAL S, Munasinghe PE, Shindikar A, Paulin J, et al
    Down-regulation of proangiogenic microRNA-126 and microRNA-132 are early modulators of diabetic cardiac microangiopathy.
    Cardiovasc Res. 2017;113:90-101.
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    November 2016
  21. LI L, Li J, Drum BM, Chen Y, et al
    Loss of AKAP150 promotes pathological remodelling and heart failure propensity by disrupting calcium cycling and contractile reserve.
    Cardiovasc Res. 2016.
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    July 2016
  22. TAKADA S, Masaki Y, Kinugawa S, Matsumoto J, et al
    Dipeptidyl peptidase-4 inhibitor improved exercise capacity and mitochondrial biogenesis in mice with heart failure via activation of GLP-1 receptor signaling.
    Cardiovasc Res. 2016.
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    May 2016
  23. LEMCKERT FA, Bournazos A, Eckert DM, Kenzler M, et al
    Lack of MG53 in human heart precludes utility as a biomarker of myocardial injury or endogenous cardioprotective factor.
    Cardiovasc Res. 2016;110:178-87.
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  24. EERENBERG ES, Teunissen PF, van den Born BJ, Meijers JC, et al
    The role of ADAMTS13 in acute myocardial infarction: cause or consequence?
    Cardiovasc Res. 2016.
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    February 2016
  25. TRITTO I, Ambrosio G
    Why does pre-clinical success in cardioprotection fail at the bedside?
    Cardiovasc Res. 2016;109:189-90.
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    January 2016
  26. GONZALEZ-SANTAMARIA J, Villalba M, Busnadiego O, Lopez-Olaneta MM, et al
    Matrix cross-linking lysyl oxidases are induced in response to myocardial infarction and promote cardiac dysfunction.
    Cardiovasc Res. 2016;109:67-78.
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