Physiological aging and inflammation-induced cellular senescence may contribute to oligodendroglial dysfunction in MS. Windener F, Grewing L, Thomas C, Dorion MF, et al. Acta Neuropathol 2024;147:82.

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Windener F, Grewing L, Thomas C, Dorion MF, et al. Physiological aging and inflammation-induced cellular senescence may contribute to oligodendroglial dysfunction in MS. Acta Neuropathol 2024;147:82.
PMID: 38722375