SGK1 mutations in DLBCL generate hyperstable protein neoisoforms that promote AKT independence. Gao J, Sidiropoulou E, Walker I, Krupka JA, et al. Blood 2021;138:959-964.

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Amedeo Smart

Independent Medical Education

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Gao J, Sidiropoulou E, Walker I, Krupka JA, et al. SGK1 mutations in DLBCL generate hyperstable protein neoisoforms that promote AKT independence. Blood 2021;138:959-964.
PMID: 33988691